Browning Reaction

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Maillard Reaction in Medicine

            As was introduced before, the Maillard reaction between amino acids and reducing sugars were first described by Louis Maillard in 1912.  For the next few decades, effects of the Maillard reaction in food processing were extensively studied by food scientists .  In recent years, the reaction between glucose and amino acids in living organisms was recognized to have an important role in human health.  The term Advanced Glycosylation Endproducts or AGEs is then introduced to describe Maillard reaction products that form under normal physiological conditions in living organisms.  Results from recent studies have shown that AGEs are responsible for a number of medical complications among diabetic patients and the aging populations.


 
 
 

AGEs and Cataract

AGEs can accumulate on long-lived proteins in living organisms such as the crystallin proteins of the ocular lens.  Monnier and Cermai (1981) first reported that yellow-brown pigments accumulated in the lens with age and at an accelerated rate in diabetic patients.

The graph on the right showed the fluorescence-excitation spectra of bovine and human lens protein.   It indicated that the intensity of yellowish increased in the bovine lens after incubation with sugar.  Also, human cataractous lens had higher intensity than young normal lenses.

Accumulated AGEs such as pyrraline, carboxymethyllysine, and pentosidine can crosslink with proteins and eventually lead to cataract formation.
 
 
 

AGEs and Aging

        In addition to crystallin proteins in the ocular lens, AGEs were also found in other long-lived proteins such as the extracellular matrix in the brain, cartilage between joints, and renal basement membrane.  Furthermore, serum albumin, LDL, and immunoglobulins have been found to be trapped in the AGEs-protein matrix, leading to thickening of basement membrane and tissue damage as seen in aged tissue.

        Results from animal studies have shown that feeding caloric-restricted, starch-restricted, or glucose-restricted diets to animals led to decreased AGEs and lowered level of AGEs-protein matrix in diabetic animals.  The effects on normal healthy animals were not as obvious.  Further studies on the presence of AGE receptor, structure of AGE epitope, and the mechanism of collagen crosslinking are required to fully understand the effects of AGEs in aging.
 
 
 

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